The accuracy of ANOVA (Table 2) did not reveal significant main effects of group (F1,24 = 2.18, P =.11) or delay (F1,24 = 0.3, P>.10) but did indicate a significant trial type main effect (F3,72 = 4.6, P =.005), which was moderated by a group × trial type interaction (F3,72 = 3.49, P =.02). As predicted, planned contrasts indicated that this interaction reflected (1) worse performance on B-X trials (t24 = 2.65, P =.01) and (2) better performance on A-Y trials for patients (t24 = 2.96, P =.007); and (3) no B-Y differences (t24 = 0.92, P =.37). The RT ANOVA (Table 2) indicated main effects of group (F1,24 = 6.5, P =.02; patients slower than controls), delay (F1,24 = 8.9, P =.006; long delay slower), and trial type (F3,72 = 26.8, P<.001). Again, the trial type main effect was moderated by a group × trial type interaction (F3,72 = 3.8, P<.01). Planned contrasts indicated that this interaction reflected (1) slower B-X RTs for patients (t24 = 2.2, P =.037), (2) no significant differences on A-Y RTs (t24 = 1.6, P =.12), (3) slower B-X than A-Y RTs in patients (t24 = 2.6, P =.016), and (4) slower A-Y than B-X RTs in controls (t24 = 2.6, P =.47). The group × trial type × delay interactions for accuracy and RT did not reach significance. However, we did find the predicted interaction with delay in d′-context (Table 2). The d′-context ANOVA indicated main effects of group (F1,24 = 5.4, P =.029) and delay (F1,24 = 18.1, P<.001) and a group × delay interaction (F1,24 = 4.1, P =.05). Planned contrasts indicated no significant differences between patients and controls at the short delay (t24 = 1.0, P =.33) but significantly decreased d′-context among patients at the long delay (t24 = 3.2, P =.004).