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Cognitive Development: Breast-Milk Benefit vs Infant Formula Hazard

Jerome L. Sullivan, MD, PhD
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Copyright 2008 American Medical Association. All Rights Reserved. Applicable FARS/DFARS Restrictions Apply to Government Use.

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Arch Gen Psychiatry. 2008;65(12):1456-1456. doi:10.1001/archpsyc.65.12.1456-a
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Kramer et al1 present evidence that breastfeeding improves children's cognitive development. They discuss 2 classes of explanation for their findings, noting that the superior cognitive development of breastfed children may be due either “to some constituent of breast milk” that promotes improved development or “to the physical and social interactions inherent in breastfeeding.”1 (p582)

There is a third class of possible explanations not addressed in the discussion. Some property of infant formula may diminish developmental potential. This alternative explanation raises the possibility that breastfed children represent a normal reference population and that formula-fed children are harmed in some way in comparison with the breastfed group. The findings appear equally compatible with either an improved outcome from breastfeeding or, alternatively, a deficit conferred by some property of infant formula.

One candidate mechanism of harm in formula-fed infants is iron-mediated subclinical brain injury. Formula may increase infant exposure to non–protein-bound iron. Apolactoferrin derived from breast milk may protect infants from early iron-mediated brain injury that would otherwise impair later neurodevelopment. A general protective effect of apolactoferrin of maternal origin was previously proposed.2 It was suggested that “apolactoferrin may be absorbed from [breast] milk in amounts sufficient to boost plasma antioxidant activity.”2 (p1344) Subsequent investigations have provided evidence that iron-mediated free radical formation is involved in brain damage in neonates3 and that non–protein bound iron is a highly significant early predictor of later neurodevelopmental outcomes.4 It has been shown that recombinant human lactoferrin added to formula or human milk attenuates, and iron present in infant formula increases, iron-mediated free radical formation and lipid peroxidation.5 There is direct evidence of an antioxidant action of human milk.6

AUTHOR INFORMATION

Correspondence: Dr Sullivan, Burnett School of Biomedical Sciences, University of Central Florida, 4475 Old Bear Run, Winter Park, FL 32792 (jlsullivan3@gmail.com).

Financial Disclosure: None reported.

REFERENCES

Kramer  MS, Aboud  F, Mironova  E, Vanilovich  I, Platt  RW, Matush  L, Igumnov  S, Fombonne  E, Bogdanovich  N, Ducruet  T, Collet  JP, Chalmers  B, Hodnett  E, Davidovsky  S, Skugarevsky  O, Trofimovich  O, Kozlova  L, Shapiro  S.Promotion of Breastfeeding Intervention Trial (PROBIT) Study Group,  Breastfeeding and child cognitive development: new evidence from a large randomized trial. Arch Gen Psychiatry 2008;65 (5) 578- 584
PubMed
Sullivan  JL. Iron, plasma antioxidants, and the oxygen radical disease of prematurity. Am J Dis Child 1988;142 (12) 1341- 1344
PubMed
Buonocore  G, Perrone  S, Bracci  R. Free radicals and brain damage in the newborn. Biol Neonate 2001;79 (3-4) 180- 186
PubMed
Buonocore  G, Perrone  S, Longini  M, Paffetti  P, Vezzosi  P, Gatti  MG, Bracci  R. Non protein bound iron as early predictive marker of neonatal brain damage. Brain 2003;126 (pt 5) 1224- 1230
PubMed
Raghuveer  TS, McGuire  EM, Martin  SM, Wagner  BA, Rebouché  CJ, Buettner  GR, Widness  JA. Lactoferrin in the preterm infants' diet attenuates iron-induced oxidation products. Pediatr Res 2002;52 (6) 964- 972
PubMed
Shoji  H, Shimizu  T, Shinohara  K, Oguchi  S, Shiga  S, Yamashiro  Y. Suppressive effects of breast milk on oxidative DNA damage in very low birthweight infants. Arch Dis Child Fetal Neonatal Ed 2004;89 (2) F136- F138
PubMed

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Country-Specific Mortality and Growth Failure in Infancy and Yound Children and Association With Material Stature

Use interactive graphics and maps to view and sort country-specific infant and early dhildhood mortality and growth failure data and their association with maternal

Kramer  MS, Aboud  F, Mironova  E, Vanilovich  I, Platt  RW, Matush  L, Igumnov  S, Fombonne  E, Bogdanovich  N, Ducruet  T, Collet  JP, Chalmers  B, Hodnett  E, Davidovsky  S, Skugarevsky  O, Trofimovich  O, Kozlova  L, Shapiro  S.Promotion of Breastfeeding Intervention Trial (PROBIT) Study Group,  Breastfeeding and child cognitive development: new evidence from a large randomized trial. Arch Gen Psychiatry 2008;65 (5) 578- 584
PubMed
Sullivan  JL. Iron, plasma antioxidants, and the oxygen radical disease of prematurity. Am J Dis Child 1988;142 (12) 1341- 1344
PubMed
Buonocore  G, Perrone  S, Bracci  R. Free radicals and brain damage in the newborn. Biol Neonate 2001;79 (3-4) 180- 186
PubMed
Buonocore  G, Perrone  S, Longini  M, Paffetti  P, Vezzosi  P, Gatti  MG, Bracci  R. Non protein bound iron as early predictive marker of neonatal brain damage. Brain 2003;126 (pt 5) 1224- 1230
PubMed
Raghuveer  TS, McGuire  EM, Martin  SM, Wagner  BA, Rebouché  CJ, Buettner  GR, Widness  JA. Lactoferrin in the preterm infants' diet attenuates iron-induced oxidation products. Pediatr Res 2002;52 (6) 964- 972
PubMed
Shoji  H, Shimizu  T, Shinohara  K, Oguchi  S, Shiga  S, Yamashiro  Y. Suppressive effects of breast milk on oxidative DNA damage in very low birthweight infants. Arch Dis Child Fetal Neonatal Ed 2004;89 (2) F136- F138
PubMed

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